We were asking
Where along each organism's development does the same founder-level intervention lose leverage over later morphology?
At nine ages, each state received one forced founder intervention and then twelve passages of release, allowing the spread among four matched founder futures to measure how redirectable the body remained.
Where along each organism's development does the same founder-level intervention lose leverage over later morphology?
Causal susceptibility stayed on a plateau and then fell by about fourteen percent just after the structural information turn, with fixed replication splits pointing the same way.
Can a disjoint fresh cohort reproduce the full age profile and distinguish falling leverage from narrowing basin width?